Evidence base

The research behind our protocols.

We reference peer-reviewed studies and preclinical research. We do not make treatment claims.

Important: The studies referenced below are provided for informational purposes only. Compounded peptides are not FDA-approved drugs. These studies do not constitute medical advice and do not imply that Elyvate's compounded formulations will produce the same results as those described in the research.

Recovery

BPC-157TB-500

BPC-157 and TB-500 are among the most studied peptides in preclinical musculoskeletal and tissue repair research. Human clinical trial data is limited — the studies below are primarily preclinical animal models.

5 referenced studies

BPC-157Informational only — not a treatment claim

The promoting effect of pentadecapeptide BPC 157 on tendon healing involves tendon outgrowth, cell survival, and cell migration

Chang CH, et al. · Journal of Applied Physiology, 2011 · PMID 21030672

Demonstrates BPC-157 accelerating tendon fibroblast migration and survival in Achilles tendon models, with dose-dependent effects on cell movement.

BPC-157Informational only — not a treatment claim

BPC 157 and Standard Angiogenic Growth Factors. Gastrointestinal Tract Healing, Lessons from Tendon, Ligament, Muscle and Bone Healing

Sikiric P, et al. · Current Pharmaceutical Design, 2018 · PMID 29998800

Reviews BPC-157's role in angiogenesis and tissue healing across multiple tissue types including gastrointestinal, tendon, and muscle.

BPC-157Informational only — not a treatment claim

Stable Gastric Pentadecapeptide BPC 157 as a Therapy for the Disable Myotendinous Junctions in Rats

Japjec M, et al. · Biomedicines, 2021 · PMID 34829776

Documents BPC-157 restoring myotendinous junction function and reversing muscle atrophy in injury models.

TB-500Informational only — not a treatment claim

Thymosin beta4: a multi-functional regenerative peptide. Basic properties and clinical applications

Goldstein AL, et al. · Expert Opinion on Biological Therapy, 2012 · PMID 22074294

Comprehensive review of thymosin beta-4 biological activities including wound healing, cell migration, anti-inflammatory effects, and clinical application potential.

TB-500Informational only — not a treatment claim

Thymosin beta4 and cardiac repair

Shrivastava S, et al. · Annals of the New York Academy of Sciences, 2010 · PMID 20536454

Demonstrates thymosin beta-4 inhibiting myocardial cell death, stimulating vessel growth, and activating cardiac progenitor cells in injury models.

Performance

CJC-1295Ipamorelin

CJC-1295 and Ipamorelin are among the most studied growth hormone secretagogues, with human clinical trial data including the studies below.

3 referenced studies

CJC-1295Informational only — not a treatment claim

Pulsatile secretion of growth hormone (GH) persists during continuous stimulation by CJC-1295, a long-acting GH-releasing hormone analog

Ionescu M, Frohman LA. · Journal of Clinical Endocrinology and Metabolism, 2006 · PMID 16352683

Human study showing CJC-1295 maintains pulsatile GH secretion patterns while significantly elevating IGF-1 levels over extended periods.

CJC-1295Informational only — not a treatment claim

Prolonged stimulation of growth hormone and insulin-like growth factor I secretion by CJC-1295, a long-acting analog of growth hormone-releasing hormone, in healthy adults

Teichman SL, et al. · Journal of Clinical Endocrinology and Metabolism, 2006 · PMID 16849409

Human clinical trial documenting sustained GH and IGF-1 elevation over multiple days following CJC-1295 administration in healthy adults.

IpamorelinInformational only — not a treatment claim

Ipamorelin, the first selective growth hormone secretagogue

Raun K, et al. · European Journal of Endocrinology, 1998 · PMID 9849822

Establishes ipamorelin as a selective GH secretagogue with minimal effects on cortisol and prolactin — a key differentiator from earlier secretagogues.

Metabolic

AOD-9604Ipamorelin

AOD-9604 is a synthetic fragment of human growth hormone studied for its selective lipolytic effects. The studies below are primarily preclinical. AOD-9604 completed Phase II clinical trials but was discontinued for lack of sufficient weight-loss efficacy at clinical endpoints.

2 referenced studies

AOD-9604Informational only — not a treatment claim

The effects of human GH and its lipolytic fragment (AOD9604) on lipid metabolism following chronic treatment in obese mice and beta3-AR knock-out mice

Heffernan MA, et al. · Endocrinology, 2001 · PMID 11713213

Demonstrates AOD-9604 reducing body fat in obese animal models through beta-3 adrenergic receptor pathways without affecting blood glucose or IGF-1 levels.

AOD-9604Informational only — not a treatment claim

Metabolic studies of a synthetic lipolytic domain (AOD9604) of human growth hormone

Ng FM, et al. · Hormone Research, 2000 · PMID 11146367

Characterizes AOD-9604 as a GH fragment that stimulates fat breakdown while showing no significant effect on blood sugar or insulin sensitivity.

Regenerative

GHK-CuKPV

GHK-Cu is a naturally occurring human tripeptide first identified in 1973. KPV is a tripeptide derived from alpha-MSH. Both have extensive preclinical literature. Human data is limited primarily to topical applications for GHK-Cu.

5 referenced studies

GHK-CuInformational only — not a treatment claim

Regenerative and Protective Actions of the GHK-Cu Peptide in the Light of the New Gene Data

Pickart L, Margolina A. · International Journal of Molecular Sciences, 2018 · PMID 29986520

Reviews GHK-Cu's role in activating genes associated with tissue repair, collagen synthesis, antioxidant defense, and anti-inflammatory signaling across more than 4,000 human genes modulated.

GHK-CuInformational only — not a treatment claim

GHK Peptide as a Natural Modulator of Multiple Cellular Pathways in Skin Regeneration

Pickart L, et al. · BioMed Research International, 2015 · PMID 26236730

Examines GHK-Cu's role in skin regeneration pathways, documenting its effects on collagen synthesis, metalloproteinase activity, and cellular remodeling.

GHK-CuInformational only — not a treatment claim

Stimulation of collagen synthesis in fibroblast cultures by the tripeptide-copper complex glycyl-L-histidyl-L-lysine-Cu2+

Maquart FX, et al. · FEBS Letters, 1988 · PMID 3169264

Original study documenting GHK-Cu stimulating collagen synthesis in human fibroblast cultures at nanomolar concentrations — the foundational collagen research for this peptide.

KPVInformational only — not a treatment claim

PepT1-Mediated Tripeptide KPV Uptake Reduces Intestinal Inflammation

Dalmasso G, et al. · Gastroenterology, 2008 · PMID 18061177

Demonstrates KPV inhibiting NF-kB and MAP kinase inflammatory pathways at nanomolar concentrations, with oral administration reducing colitis incidence in animal models.

KPVInformational only — not a treatment claim

Orally Targeted Delivery of Tripeptide KPV via Hyaluronic Acid-Functionalized Nanoparticles Efficiently Alleviates Ulcerative Colitis

Xiao B, et al. · Molecular Therapy, 2017 · PMID 28143741

Shows KPV reducing intestinal inflammation and accelerating mucosal healing in colitis models, with targeted nanoparticle delivery increasing efficacy 12,000-fold over free solution.

Cognitive

SelankSemax

Selank and Semax were developed by the Russian Academy of Sciences. Semax is approved in Russia for stroke and neurological conditions. Most studies are from Russian clinical and preclinical research. Large-scale Western randomized controlled trials are limited.

3 referenced studies

SelankInformational only — not a treatment claim

Efficacy and possible mechanisms of action of a new peptide anxiolytic selank in the therapy of generalized anxiety disorders and neurasthenia

Zozulia AA, et al. · Zhurnal Nevrologii i Psikhiatrii, 2008 · PMID 18454096

Clinical study of 62 patients showing Selank produced anxiolytic effects comparable to medazepam while also demonstrating antiasthenic and psychostimulant effects without sedation.

SelankInformational only — not a treatment claim

Functional Connectomic Approach to Studying Selank and Semax Effects

Melik-Kasumov T, et al. · Frontiers in Neuroscience, 2020 · PMID 32342318

fMRI study in 52 healthy participants documenting distinct effects of Selank and Semax on functional connectivity between the amygdala and temporal cortex regions involved in anxiety regulation.

SemaxInformational only — not a treatment claim

Semax, an analog of ACTH(4-10) with cognitive effects, regulates BDNF and trkB expression in the rat hippocampus

Dolotov OV, et al. · Brain Research, 2006 · PMID 16996037

Documents Semax producing a 1.4-fold increase in hippocampal BDNF protein and 3-fold increase in BDNF mRNA, with treated animals showing improved conditioned avoidance learning.

A note on evidence levels

The majority of peptide research cited on this page comes from preclinical animal studies. Human clinical trial data is limited for most of these compounds outside of CJC-1295, Ipamorelin, and Semax. Elyvate does not claim these studies prove efficacy in humans. Our physicians review each intake individually and prescribe based on their clinical judgment, not on marketing claims.

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